langendorff apparatus model iph-w2 (Primetech Corporation)
90
Structured Review
Primetech Corporation
langendorff apparatus model iph-w2
Langendorff Apparatus Model Iph W2, supplied by Primetech Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/langendorff+apparatus+model+iph-w2/langendorff+apparatus+model+iph+w2/pm32683442-69-15-19
Average 90 stars, based on 1 article reviews
Langendorff Apparatus Model Iph W2, supplied by Primetech Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/langendorff+apparatus+model+iph-w2/langendorff+apparatus+model+iph+w2/pm32683442-69-15-19
Average 90 stars, based on 1 article reviews
langendorff apparatus model iph-w2 - by Bioz Stars,
2026-09
90/100 stars
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Related Articles
other:Article Title: Important Role of Endothelial Caveolin-1 in the Protective Role of Endothelium-dependent Hyperpolarization Against Nitric Oxide–Mediated Nitrative Stress in Microcirculation in Mice Article Snippet: 1 2 Aims: Nitric oxide (NO) and endothelium-dependent hyperpolarization (EDH) play important 3 roles in maintaining cardiovascular homeostasis.. We have previously demonstrated that 4 endothelial NO synthase (eNOS) plays diverse roles depending on vessel size, as a NO 5 generating system in conduit arteries and an EDH-mediated system in resistance arteries, for 6 which caveolin-1(Cav-1) is involved.. However, the physiological role of endothelial Cav-1 in 7 microvessels remains to be elucidated. Article Title: Disruption of Physiological Balance Between Nitric Oxide and Endothelium-Dependent Hyperpolarization Impairs Cardiovascular Homeostasis in Mice Article Snippet: Then, the canulated heart was mounted onto a Langendorff apparatus (Model IPH-W2, Article Title: Low-intensity pulsed ultrasound ameliorates cardiac diastolic dysfunction in mice: a possible novel therapy for heart failure with preserved left ventricular ejection fraction. Article Snippet: Department of Cardiovascular Medicine, Tohoku University Graduate School of Medicine, 1-1 Seiryo-machi, Aoba-ku, Sendai 980-8574, Japan; Department of Evidence-Based Cardiovascular Medicine, Tohoku University Graduate School of Medicine, Sendai, Japan; Department of Clinical Physiology, Tohoku University Graduate School of Medicine, Sendai, Japan; Department of Electronic Engineering, Tohoku University Graduate School of Engineering, Sendai, Japan; and Division of Biomedical Measurements and Diagnostics, Tohoku University Graduate School of Biomedical Engineering, Sendai, Japan |